Neuroinflammation Mechanisms and Management 2nd Edition by Paul L. Wood – Ebook PDF Instant Download/Delivery: 978-158829002, 11588290026
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Product details:
ISBN 10: 11588290026
ISBN 13: 978-158829002
Author: Paul L. Wood
Table of contents:
I. Neuroinflammatory Mechanisms
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Microglia: Roles of Microglia in Chronic Neurodegenerative Diseases
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Apoptosis vs Nonapoptotic Mechanisms in Neurodegeneration
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Role(s) of Mitogen and Stress-Activated Kinases in Neurodegeneration
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Roles of Chemokines and Their Receptors in Neuroinflammation
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Neurotoxic Mechanisms of Nitric Oxide
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Chronic Intracerebral LPS as a Model of Neuroinflammation
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Peroxisome Proliferator-Activated Receptor Gamma Agonists: Potential Therapeutic Agents for Neuroinflammation
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Neuroinflammation-Mediated Neurotoxin Production in Neurodegenerative Diseases: Potential of Nitrones as Therapeutics
II. Stroke and TBI
9. Inflammation and Potential Anti-Inflammatory Approaches in Stroke
10. Neuroinflammation as an Important Pathogenic Mechanism in Spinal Cord Injury
11. Type IV Collagenases and Blood-Brain Barrier Breakdown in Brain Ischemia
III. Alzheimer’s Disease
12. Neuroinflammatory Environments Promote Amyloid-β Deposition and Posttranslational Modification
13. Microglial Responses in Alzheimer’s Disease: Recent Studies in Transgenic Mice and Alzheimer’s Disease Brains
14. The Amyloid Hypothesis of Cognitive Dysfunction
15. The Cerebellum in AD: A Case for Arrested Neuroinflammation?
16. The Neuroinflammatory Components of the Trimethyltin (TMT) Model of Hippocampal Neurodegeneration
17. Inflammation and Cyclo-Oxygenase in Alzheimer’s Disease: Experimental Approaches and Therapeutic Implications
IV. Multiple Sclerosis
18. Experimental Autoimmune Encephalomyelitis
19. Neuroimmunologic Mechanisms in the Etiology of Multiple Sclerosis
20. In Vivo Imaging of Neuroinflammation in Neurodegenerative Diseases
V. Parkinson’s and Huntington’s Diseases
21. Inflammatory Mechanisms in Parkinson’s Disease
22. Neuroinflammatory Components of the 3-Nitropropionic Acid Model of Striatal Neurodegeneration
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